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Viruses that prove common descent

pshun2404

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Barry...for a great example, I would say MANY (not all) of your camp accept the work of Theobald in confirming a UCA, but when his process and procedures were analyzed by other equally reliable scientists they were not so convinced.

In the "Scientific World Journal", (2012), Article ID 479824, the authors (Yonzawa and Hasegawa) point out a problem with the earlier research of Theobald (Nature 465, 219–222 (2010)), who claimed his research confirmed the idea of a Universal Common Ancestor. Their analysis and review of his work led them to the following conclusion.

The most serious problem of Theobald’s analysis is that he used aligned sequences compiled by Brown et al. [1], who were interested in resolving the phylogenetic relationships among archaebacteria, eubacteria, and eukaryotes, including whether each domain of life constitutes a monophyletic clade. So they a priory assumed the existence of UCA. Indeed, alignment is a procedure based on an assumption that the sequences have diverged from a common ancestral sequence. Brown et al. wrote “Individual protein families were first computer aligned and then we manually refined the alignments. We removed poorly conserved regions in individual protein alignments.” This procedure clearly assumes the existence of UCA, and this was not a problem for Brown et al., because what they were interested in was the phylogenetic relationship among all species on Earth, and the existence of UCA was supported by circumstantial evidence . However, in proving the existence of UCA, the alignment procedure should not be used, because it gives a strong bias for the UCA hypothesis.”

It appears in Theobald’s research, using humanly programmed computer assumptions and manipulating other factors, was able to make the preconceived conclusion (UCA) look like it had been confirmed. When people pushing this belief do this, it should make all who have swallowed the notion ask questions and become suspect. When taught or used as support these facts should be revealed up front (especially in school when being taught).

So first Theobald based his model on Browns who admitting "Individual protein families were first computer aligned, and then we manually refined the alignments. We removed poorly conserved regions in individual protein alignments.'

Which reveals

a) they used a computer program to align similar areas (intelligently designed by someone already convinced the hypothesis is true) and then
b) manually REFINED the alignments (because they did not actually match up), then finally
c) removed areas they declared "poorly conserved"

Now you can call this work astounding if you would like, but I call it the result of intelligent design (necessary components of most scientific endeavor...which is fine just admit it and separate the real form the devised results) and we must not exclude the FACT that they tweaked the data to get the DESIRED result (which already was presupposed to be true).

So why would anybody with any semblance of intellectual integrity blind themselves to the role assumption plays in many of these conclusions?
 
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pshun2404

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Barry...you said,

"A gullible reader will assume that you are quoting from the paper"

I don't know how you do it, but when I quote someone I use what are called "quotation marks"! Only a dummy would try and make it appear as if I am trying to make this a quotation (or accuse me of such). And by the way, your Veritas stories are replete with much of the same exact thing (only I can see the difference clearly, as you did here), where as others voewing your presentations might think all you present 'matter of factly' actually are (and we both know what a mistake that could be when viewing opinion based paragraphs in anyone's presentations).
 
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xianghua

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Sequences that differentiate cell types would necessarily need to be transcribed. It's very difficult to see what you think your point is here.

the point is that we found a good correlation between complexity and the amount of "junk". so the more junk you have -then you will be more complex. how do you explain this correlation?

Good luck with proving that nature was designed, but that's irrelevant. Retroviruses and the ERVs they create prove common descent, whether they were designed or not.

its very relevant. because the erv argument base about belief- that those are indeed a viral insertions and that they have a great space to insert the genome. but if we have a proof that nature was designed- then we dont need those beliefs.
 
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xianghua

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Or used to be functional.

The question seems to be how the ervs got there. How would just being functional mean they got there a certain way?
because if the genome is about 100% functional (just for the sake of the argument) then retrovirus cant insert (and get fixed) in any place, only in a few places in the genome if any. and indeed: a lots of ervs are also species specific. some of them are even shared in far species but not in the closer species (see above pshun comment).
 
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dad

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because if the genome is about 100% functional (just for the sake of the argument) then retrovirus cant insert (and get fixed) in any place, only in a few places in the genome if any. and indeed: a lots of ervs are also species specific. some of them are even shared in far species but not in the closer species (see above pshun comment).
Since they did insert, I guess you think they were functional. That is all well and good in the present. However in the past, nature may have been different, so present ways and reasons for things have no value. In other words ancient ervs may have transferred in a way we now do not know about, and that can't happen anymore.
 
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the point is that we found a good correlation between complexity and the amount of "junk". so the more junk you have -then you will be more complex. how do you explain this correlation?



its very relevant. because the erv argument base about belief- that those are indeed a viral insertions and that they have a great space to insert the genome. but if we have a proof that nature was designed- then we dont need those beliefs.
As I said, good luck with proving that nature was designed. ;)
 
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pshun2404

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Nice article YOU wrote on YOUR opinions regarding creationism, mixed into YOUR article about the subject matter at hand, but what does that have to do with the price of rice? Should have just left the scientific opinion, it was good stuff, but it also assumes the UCA as did the researchers going in.

Now in the CERV 30 chart you provided, this data could just as easily be interpreted to mean that this human's totally human ancestor (not even all humans), was infected at this site at one time, while the chimp sequences exhibited this possible insertion being at two places, and their chimp ancestor could have been infected with the same virus on two entirely different occasions.

So it COULD BE that their analysis is correct, but they already were looking for examples of similarity (because they already believe the UCA hypothesis) which adds a degree of bias in their interpretation. However, it COULD also be, that seen just as it is, it does not sufficiently demonstrate their conclusion.

You CAN see that right? I mean you do not have to agree with the alternative explanation in this case, but you can see the possibility of it, right?

Many better examples were included in the more information here link but I have no doubt many ERVs ARE ERVs...but even these do not prove common descent.
 
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Nice article YOU wrote on YOUR opinions regarding creationism, mixed into YOUR article about the subject matter at hand, but what does that have to do with the price of rice?
Not my article, but it is about ERVs, which is the subject of this discussion.
 
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pshun2404

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In your 'more info here' link on indels? The first example I am afraid is not a good one. Yes there appears to be an insertion in the first line of human DNA (which is not present in the chimp) but IS IT an insertion or just a difference in the two genomes in general, and the alleged chimp deletion may never even have ever been a part of the chimp's sequence in the first place. While there are examples of actual apparent insertions or deletions (like in the second example), this first one is definitely NOT one of them (it is interpretation, i.e., opinion) unless you can show early chimp genomes where it is present and now gone, and even then it does not prove common descent, just that it is an actual (not supposed) deletion.

Good article nonetheless so thanks.
 
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pshun2404

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Not my article, but it is about ERVs, which is the subject of this discussion.

I stand corrected on it being one of yours (I found it very similar in style and tone), but they must be like an answersingenesis rag for the other side (otherwise why go there?).
 
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Many better examples were included in the more information here link but I have no doubt many ERVs ARE ERVs...but even these do not prove common descent.
I know I am repeating myself, but it appears to be necessary. The vast majority of ERV and ERV remnants that can be found in human and chimp genomes are to be found in precisely corresponding locations in the DNA of each species. As the integrase enzyme that - um - integrates retroviral DNA into the host genome cannot target specific loci, to believe that the commonality of location among all these elements is a coincidence
is beyond the bounds of credibility. That a relatively small number of ERV elements are unique to chimps or unique to humans can be easily explained by post-speciation endogenizations, or (less easy to comprehend but nevertheless a real phenomenon), incomplete lineage sorting where a preintegration site goes to fixation in one species, but the postintegration site goes to fixation in the other.

And here again is my link that explains how we can be confident that an ERV is the inherited result of a retroviral integration. Veritas: ERV FAQ: Why do virologists and geneticists think that ERVs come from retroviruses? Isn't that just supposition on their part?
 
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xianghua

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Or used to be functional.

The question seems to be how the ervs got there. How would just being functional mean they got there a certain way?
even if they are non-functional the claim will be valid. because their insertions are far from random.
 
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even if they are non-functional the claim will be valid. because their insertions are far from random.
Like road traffic accidents are non-random. They tend to occur more frequently in accident blackspots. But each RTA is not precisely the same, involving the same vehicles at exactly the same spot. I've shown you surveys of integration sites and explained the statistical nature of site "preferences". Stop wasting my time.
 
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xianghua

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Please ignore. (Can't see how to delete.)

Like road traffic accidents are non-random. They tend to occur more frequently in accident blackspots. But each RTA is not precisely the same, involving the same vehicles at exactly the same spot. I've shown you surveys of integration sites and explained the statistical nature of site "preferences". Stop wasting my time.
simple question: if the genome is about 100 functional, how many ervs can be add to the genome and be fixed?
 
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