I guess the OP got lost somewhere.
Somewhere in AV's street, as usual
However, getting back to ERV's. We share genetic markers with many species in the plant and animal world, but this doesn't prove we are related; or the common ancestor, because similarity of function from one organism to another is not in itself evidence of common descent.
What
is the function of an endogenous retrovirus? Or of a pseudogene, for that matter.
ERV's insert information into the host chromosome, but this insertion is not proof of creating new information.
What does information have to do with common descent?
True, ERV's change the genetic structure, but the problem with this is the definition of the term evolution has been defined to mean 'any' change in a species.
Why is that a problem?
Since ERV's add information, either by insertion or reinfection, this qualifies as change, and thus, I guess, evolution.
When a gene becomes a pseudogene (or a retrovirus an inert ERV) it's as much evolution as the appearance of nylonases. Evolution is not defined with a change of information (and I don't see why it should be)
HIV - 1 is a retrovirus. Does it cause the infected to 'evolve'? Of course not, but which part of the question is the OP after?
Of course yes (if by "the infected" we mean the population of potential hosts). It's a selective pressure like any other pathogen. But that's irrelevant to the point about ERVs.
Is it that the mere existence of ERV's proves evolution, or that when inserted ERV's create new information and are thus proof of evolution?
No, it's the particular distribution of ERVs in various genomes. For an endogenous retrovirus to be found at a certain locus in a whole species or larger group it once had to (1) infect germ cells (2) infect a germ cell
that later becomes an organism (3) insert
at random in that particular place (and a mammalian genome, as you know, is big) (4) get fixed in the population (either by some weird advantage it provides or by genetic drift).
The chance of two species sharing the same ERV at the same locus
independently is rather small. And it's not a single ERV we are talking about, it's hundreds of thousands distributed in a very suggestive pattern.
In addition, how many times has non-coding DNA created a new functional protien? This would be creating new information.
Why does it have to be non-coding DNA? (I still don't understand how "information" comes into this, btw)