Regarding this current discussion, your claim that one must first measure the amount of information contained in the gene pool is in this case anyway, a classical example of bifurcation (false dilemma). That is, whether intentional or not, you have still evaded the stock issues previously revealed in this discussion.
Lanakila, I havent evaded anything. Your premise is that evolution cannot increase information. This has two obvious flaws.
1. An unsupported assumption that the amount of information is related to macroevolutionary changes. (I assume her that you are trying to argue against macroevolution, since youve said elsewhere that you have no problem with microevolution.)
2. You havent actually shown that it is impossible for evolution to increase information. You have claimed as such but given no proof that has withstood scrutiny.
1. Natural selection manipulates pre-existing information.
2. Genetic mutation deletes/rearranges pre-existing information.
3. Ergo: the theory of biological evolution cannot account for the pre-existing information.
Three does not follow from One and Two because the theory of evolution is more than mutation plus natural selection. Also one and two are still suffering from the same flaw: a direct linking of information theory to genetics and evolution. You have said that the genome contains information. But what type? Fisher? Shannon? Kolmogorov?
To be sure, the very best natural selection can do with a mutation is to produce what is termed balanced polymorphism like sickle cell anemia. In this case, this condition caused by a genetic mutation provides resistance to malaria, but likewise threatens the overall heath/survivability of the organism.
Sickle cell anemia is not a balanced polymorphism. Its a genetic disease caused by being homozygous for the Hemoglobin-S allele at the Beta-Hemoglobin locus. In parts of the world where malaria occurs, selection does currently maintain polymorphism at that locus because heterozygous individuals have the greatest fitness. However, there is also another allele Hemoglobin-C, which provides the advantages of the Hemoglobin-S allele with none of the drawbacks. Scientists predict that, if current conditions stay the same, the Hemoglobin-C allele will eventually become fixed in malaria-infected populations in Africa.
Furthermore, polymorphic loci are very important to the preservation of genetic diversity. Evolution occurs by both the increase and decrease of genetic diversity. Natural selection operates on the level of the individual not its genes. Selection does not act to promote heterozygous loci, it only acts to promote phenotypes that are important to success. Natural selection does not get to look at the genotype.
The question is not how much information is there, in the gene pool, but rather where did the information come from in the first place!
Assuming by information you are referring to this gene and its alleles, then they got there by mutation. Hemoglobin-S is simply a mutation of the wildtype allele. A point mutation causes the amino acid valine to replace a glutamine. One nucleotide difference is enough to determine whether an individual dies from malaria or not. If youre wondering where the locus came from, the answer is mutation too. In this instance the mutation was a gene duplication of another globin locus. This duplication increased the amount of information in the genome for coding a globin protein. At first the information was redundant, but subsequent mutations changed the genes so much that they contained information for different proteins. It is not hard to see how the accumulation of mutations can change and add information for new proteins into the genome. Another globin duplication-divergence event, which produced the fetal hemoglobins, was extremely important in our evolutionary history because it allowed fetuses to have a higher affinity to oxygen than adults. Thats very important to the transmission of oxygen in the placenta.
See
this page for more discussion on the evolution of globin genes.
The various functions of genetic mutations (insertion, translocation, deletion, and inversion) are in fact, merely deletion, rearrangement or a combination of the same of pre-existing information this is inescapable.
See above. Pre-existing information is initially copied, but subsequent mutations can also change it to make it something novel and in turn add information for novel proteins and phenotypes.
The fact is that neo-Darwinistic theory is intrinsically theory laden and a convoluted representation of both scientific data and philosophical foundations of cosmogony.
Lanakila, biological evolution has no more to do with the origins of the universe or life than does music theory. Maybe you should stop learning about evolution from creationist sources. After all, do you ask Castro to explain capitalism to you?
Like it or not, you have quite a dilemma my friends, because classical evolutionary theory unequivocally begins with a presupposition (i.e. faith) that the worldview of naturalism is valid and then proceeds to evaluate the scientific data accordingly. Yet, the theoretical infrastructure of such cannot support the scientific evidence or philosophical data.
I beg of you, please attempt to conduct a careful introspective evaluation, of your hearts and minds, to make sure that you are seeking after truth, regardless of the consequences. It is obligatory to us within the academic community to be honest, with both ourselves and the data, so that we achieve not our conclusion, but the conclusionobjective reality.
Like it or not, Lanakila, evolutionary biology is a science. Its accuracy cannot be determined by whatever philosophical, emotional, religious, and political arguments and conclusions people might draw.