In the Italian village of Limone sul Garda, there is a family with a mutation related to their HDL cholesterol which made them insusceptible to heart failure, despite their high-risk diet and lifestyle. This family is now being genetically-sampled in the hopes of treating heart disease.
Another example of new variance is the Glycophorin A somatic cell mutation (Jensen, R. H., S. Zhang, et al. (1997) which has been identified in some Tibetans, which allows them to endure prolongued periods at altitudes of 7,000 feet without succumbing to apoplexia, or altitude sickness. A different, but similar mutation was identified in high altitude natives in the Andes.
http://jeb.biologists.org/cgi/content/full/204/18/3151
Another example of that is the CCR5-delta 32 mutation. About 10% of whites of European origin now carry it. But the incidence is only 2% in central Asia, and is completely absent among East Asians, Africans, and American Indians. It appears to have suddenly become relatively common among white Europeans about 700 years ago, evidently as a result of the Black Plague, indicating another example of natural selection allowing one gene dominance in a changing environment. It is harmless (or neutral) in every respect other than its one clearly beneficial feature; if one inherits this gene from both parents, they will be especially resistant (if not immune) to AIDS.
(source: Science-Frontiers.com / PBS.org)
For another example, weve also identified an emerging population of tetrachromatic women who can see a bit of the normally invisible ultraviolet spectrum.
http://www.guardian.co.uk/Archive/Article/0,4273,4128183,00.html
Theres also a family in Germany who are already unusually strong. But in one case, a child was born with a double copy of an anti-myostatin mutation carried by both parents. The result is a herculian kiddo who was examined at only a few days old for his unusually well-developed muscles. By four years old, he had twice the mucle mass of normal children, and half the fat. Pharmaceutical synthesis of this mutation is being examined for potential use against muscular dystrophy or sarcopenia.
http://cnews.canoe.ca/CNEWS/Science/2004/06/24/512617.html
There is also a family in Connecticut that has been identified as having hyperdense, virtually unbreakable bones:
Members of this family carry a genetic mutation that causes high bone density. They have a deep and wide jaw and bony growth on the palate. Richard P. Lifton, M.D., Ph.D., chair of the Department of Genetics, along with Karl L. Insogna, M.D., professor of medicine and director of the Yale Bone Center, and colleagues, traced the mutation to a gene that was the subject of an earlier study. In that study researchers showed that low bone density could be caused by a mutation that disrupts the function of a gene called LRP5. In the recent study, the Yale team mapped the familys genetic mutation to the same chromosome segment in LRP5. It made us wonder if a different mutation increased LRP5 function, leading to an opposite phenotype, that is, high bone density, Lifton said.
Family members, according to the investigators, have bones so strong they rival those of a character in the 2000 movie Unbreakable. If there are living counterparts to the [hero] in Unbreakable, who is in a terrible train wreck and walks away without a single broken bone, theyre members of this family, said Lifton. They have extraordinarily dense bones and there is no history of fractures. These people have about the strongest bones on the entire planet.
http://info.med.yale.edu/external/pubs/ym_au02/findings.html
Do these satisfy your request? Or is your faith of the type which decides in advance that no amount of evidence or reason will ever cause you to question the absolute accuracy of your preconceived notions?
No it doesn't. Whoever told you otherwise was either ignorant or trying to deceive you deliberately.