Of course. Because it is over your head.
Did you really just confuse polymorphism with polyploidy?
Polymorphism, polyploidy, allele, allie, same thing.
No, You did is all.....
""Genetic polymorphism is the occurrence in the same population of
two or more alleles at one locus,"
"A polyploid is a cell or organism that contains more than two paired (homologous)
sets of chromosomes."
The human leukocyte antigen what?
Is your understanding of biology that weak?
https://en.wikipedia.org/wiki/Human_leukocyte_antigen
"The
human leukocyte antigen (
HLA) system or complex is a gene complex"
I'm sorry, did my saying genome instead of gene complex so throw you off that you couldn't understand?????
What is a non-functioning genome?
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4035993/
"However, the biochemically active regions cover a much larger fraction of the genome than do evolutionarily conserved regions, raising the question of whether nonconserved but biochemically active regions are truly functional."
". If repetitive DNA elements could be equated with nonfunctional DNA, then one would surmise that the human genome contains vast nonfunctional regions because nearly 50% of nucleotides in the human genome are readily recognizable as repeat elements, often of high degeneracy. Moreover, comparative genomics studies have found that only 5% of mammalian genomes are under strong evolutionary constraint across multiple species (e.g., human, mouse, and dog) (
2,
3)."
If they are now non-functional, what is your evidence that they even were once functional?
The fact that they are now highly degenerate, because of your savior mutation.
This is a mere assertion. Please for once present evidence that this is the case - you can start by providing documentation as to which "genomes" coded for which race.
If the genomes did not contain race specific points, then there would be no sense to do research on diseases more prone to one race than another. yes, i understand race is not politically correct and that biologists have bowed to political pressure, and yet they still understand some races are more prone to certain diseases than the other races. Which would not be a factor if race had no consideration in reality.
Races? No. Alleles? Obviously - the Grant paper you probably now regret ever finding says so:
"We may add one more difference between a mutated allele and one introduced by hybridization. The mutated allele has been altered randomly, whereas the one introduced by hybridization has been shaped by natural selection, albeit in a differentiated genome (deleterious mutations have been purged and any beneficial mutations gone to fixation by selection)."
That is, the newly introduced allele was shaped b y MUTATION.
Allies... allleles...
Genetic strand (remember that one?
HILARIOUS!)...
Continuous traits = mutations..
Amazing...
Yes, I understand you don't understand what is said, but only see what you want to.
"We may add one more difference between
a mutated allele and
one introduced by hybridization. The
mutated allele has been altered randomly, whereas the one [allele] introduced by hybridization has been shaped by natural selection, albeit in a differentiated genome (deleterious mutations have been purged and any beneficial mutations gone to fixation by selection)."
In other words a genome in which deleterious mutations have been purged and any beneficial mutations already gone to fixation. Therefore the new allele is not the mutated allele, all mutations have been deleted or fixed already.
This is seen in ALL studies....
https://www.nature.com/articles/ncomms13195
"Of a total of 16.6 million single-nucleotide polymorphisms (SNPs) segregating across all populations, 11.8% were shared with the American crow....Principle component analyses (
Fig. 1c), ancestral population graph reconstruction (
Fig. 1d), and phylogenetic network analyses (
Supplementary Figs 2 and 3) clearly demonstrate an independent origin of both pied subspecies,
pectoralis and
cornix, from black ancestors....Overall, this portrays an evolutionary scenario where a single
cornix population expanded into suitable habitat after the last glacial retreat and came into secondary contact with divergent black populations approximately ten millennia ago....had higher levels of genome-wide linkage disequilibrium (LD) than neighbouring parental populations (Kruskal–Wallis test,
P=0.0308, 0.0063), and showed a range of admixture proportions (0.532–0.886) attesting to ongoing backcrossing....."
"...First, genome-wide profiles of the population recombination rate
ρ (=4
Ner) and LD were positively correlated ‘among’ all possible population pairs supporting the assumption of broad-scale recombination rate conservation expected across this short evolutionary timescale; and hence fulfilling a central precondition for shared linked selection across populations. Second, consistent with a role of shared linked selection in
reducing polymorphism, nucleotide diversity
π was correlated ‘among’ populations, even when controlling for the effect of
μ (approximated by synonymous substitution rate). ‘Within’ populations,
ρ (and resulting LD) correlated strongly with
π (=4
Neμ).
With little evidence for recombination-associated mutation (and hence
r∼
μ) (ref.
15) and
ρ generally recapitulating recombination rate......
The affect of crossbreeding in a differentiated genome leads to "reducing polymorphism" and leads to little evidence or recombination-associated mutation.
Don't even pretend like only you can understand. That is the first indication of a pure hack...... which is exactly what you are....
