How is the chemical/physical process of microevolution different from macroevolution?
What prevents macroevolution from happening?
What prevents macroevolution from happening?
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Originally posted by chickenman
How is the chemical/physical process of microevolution different from macroevolution?
What prevents macroevolution from happening?
Originally posted by chickenman
I realise the difference on the macroscopic level - I want to know what the difference is at the molecular level - if one can happen and the other can't then the processes required must be different
Originally posted by chickenman
i don't think they're different seesaw, I think both have happened. Creationists draw a line between them though, and I want to know what their justification is for that line
God made the first animals perfect there gene-pool was astronomical,and still would have been pretty close to that after the flood.hers an example of humans today. first the matter in the known universe is 10 to the 80 power.the combination of variation among humans today is 10 to the 2017 power.and that is how we have the great variation among animals since the flood. Originally posted by Pete Harcoff
Macroevolution tends to be a moving target when it comes to creationists. I've found some claim that speciation can't occur. Show them examples where speciation has occurred, and they'll redefine it to claim that changes between "kinds" can't occur (whatever a "kind" is).
It gets even more fun when you start to consider the problems of modern terrestrial life on this planet being descended from a limited number of animals Noah took on his ark during the flood (assuming you're dealing with someone who takes the Bible literally). Without some level of "macroevolution", how do you explain that?
alright now first lets see what you are talking about.there are micro-increases in information but it is only like a switch thats already present that switches on and off .this is true but there has not been an increase in any new information that just miraculusly appearsOriginally posted by chickenman
increases in information have been observed and are possible
you'll have to invent something else to differentiate "macro" from "micro"
Originally posted by Freedom777
alright now first lets see what you are talking about.there are micro-increases in information but it is only like a switch thats already present that switches on and off .this is true but there has not been an increase in any new information that just miraculusly appears

i believe that is a scrambleing of information that already exists you can have say todrakrov and scramble that and you will never get a z or w or s ect...and this is where its at.Originally posted by chickenman
unequal crossing over can duplicate large stretches of DNA - this generates new information.
find something else to differentiate between macro and micro, because mutations can generate new information
Originally posted by Freedom777
i believe that is a scrambleing of information that already exists you can have say todrakrov and scramble that and you will never get a z or w or s ect...and this is where its at.
Originally posted by chickenman
okay lets do a little thought experiment
1: Eur J Hum Genet 2000 Mar;8(3):229-35
Related Articles, Links
Prenatal detection of a 17p11.2 duplication resulting from a rare recombination event and novel PCR-based strategy for molecular identification of Charcot-Marie-Tooth disease type 1A.
Bernard R, Labelle V, Negre P, Tardieu S, Azulay JP, Malzac P, Mattei JF, Leguern E, Philip N, Levy N.
Departement de Genetique Medicale, Hopital d'enfants de la Timone, Marseille, France.
Charcot-Marie-Tooth disease, type 1A (CMT1A) is caused in most cases by a 1.5 Mb duplication on chromosome 17p11.2 arising after unequal crossing-over between repeated sequences called CMT1A-REPs, flanking the 1.5 Mb unit. A 3.2 kb recombination hot spot has been defined, resulting in a junction fragment between EcoRI (distal CMT1A-REP) and SacI (proximal CMT1A-REP). This was further reduced to a 1.7kb EcoRI-NsiI fragment, and recently to a 731 bp hot spot region within this fragment. We describe the CMT1A- REPs-based PCR method used to identify CMT1A duplications and report on a family case in which a 29-year-old pregnant woman requested prenatal diagnosis for two successive pregnancies because her husband was affected with CMT1A. Our method enabled us to characterise the duplication in both foetuses and demonstrate that it arose from a rare recombination event taking place outside the 1.7 kb region. Since our approach is simple and enables the entire set of duplications occurring after recombination in the enlarged 3.2kb region including the hot spot to be detected, we suggest it might be considered for use in primary screening for pre- and postnatal diagnosis of CMT1A.
PMID: 10780790 [PubMed - indexed for MEDLINE]