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rmwilliamsll said:There are imaginable barriers between kinds, my favorite is one i've posted here several times, is a different genetic code for each kind.
What is interesting about the question- can we see a kind barrier? and your short answer-" laws of inheritance in Mendelian genetics."
what are the Mendelian genetics rules?
there is a reasonable intro at:
http://onlinetc.its.brooklyn.cuny.edu/Core81/chap8.html
The genes in what Medel called the elementum and we now know are the chromosomes. The dogma of biology is DNA and the clearest limit of evolutionary change is in the energetic cost of adaptative changes. I am somewhat familar with Genetics but thanks for the link just the same.
i don't think it is any more complex then high school f1, f2 type of exercises. How does this form a kind barrier?
The random variations that are the cause of change through random variation are fixed in the genotype and are cyclical with regards to the expressed phenotype. It is this random cross over of genes and the ability of enzymes to turn certain genes on and off that gave rise to the diversity we see spanning the natural world. Bear in mind the patterns that Mendel reduced to ratios tend to run in cycles and swing from the right to the left of a median.
next- what are your kinds? class. actually it is the first time i've seen this answer from a YECist. so what is class in systematic classification?
see:
http://www.nationmaster.com/encyclopedia/Class-(biology)
# Class
# Order
# Family
# Genus
# Species
so what are some examples of classes?
Insecta, Mammalia,
That is the first time someone actually accepted a definition of 'kinds' and didn't just ask the question again. I have a definition for class and I don't really know what object oriented biology is but it sounds like a reasonably good working definition:
"A class is a template from which object instances are created. It specifies the common characteristics that objects created from it will contain. Classes are created from gene products whose characteristics are defined by the GO (Gene Ontology) molecular function and cellular component terms. Example: The class Smad 2 is created based on the properties of the gene product Smad 2, which are defined by molecular functions such as protein homodimerization (GO:0042803) and ATP binding (GO:0042301)"
http://www.nodalpoint.org/node/1645
The class, or the kind, will depend largely on what characteristics you are attempting to classify. I don't intend to do an elaborate study on this just simply qualifying my use of the term 'kind'.
personally, i think this too large a classification for most YECists. but it's your dime.
I don't think that YEC has a standard classification system and they tend to like ambiquity when dealing with classification systems. Taxonomy is a subjective classification system that is organized largely for the sake of convienance.
so essentially your argument is that there is an absolute barrier between say mammals and birds. and that this barrier is expressed in the basic genetic laws discovered by Mendel.
Yes, of course that is what I am saying. I am also saying this is directly contradicted by neodarwinism that claims there are no such barriors. For me, the choice is between Mendel and Darwin because they both could not have been right.
There is no mechanism differences in any living creature but a few genetic code variations in archaebacteria. The only thing different between a mammal and a bird is the specific genes. The DNA is the same, evidence is the interesting fact that we have thousands of virus genes encoded into our genome. There appears to be no barrier to integration from any number of outside of mammalia order viruses.
Speaking of viruses I assume you are familiar with the Spiegelman's monster where an RNA genome of 4,500 nucleotides over 74 generations was reduced to 220 bases. I noticed this again in the Chimpanzee/human DNA comparison where they postulated that the original genome of the mrca was larger then either one dispite the large number of insertions. Creationists contend that the effect of mutations is allways a net loss of information and I tend to agree, at least in principle.
By the way, DNA is structured the same and we will be 25% identical in all living things since there are only four base combinations. So being less then 80% simular and only about 39% identical in our protein coding genes would be a logical disproof of common ancestory. What TOE lacks is a genetic basis for these macroevolutionary change and rely on accumulation of minor variations over incomprehensible periods of time. It is an absurd assumption, not a demonstrated mechanism.
this data alone, this the enormous amount of work being done shows me convincingly that there is simply no barrier to the integration of avian genes or dna into the human genome via virus infection.
Ok, that is viruses, now how about protein coding genes. What are the effects of fundamental change to these DNA stands as directly observed and empirically demonstrated in modern genetics?
now this is certainly at a level lower than your gene analysis, but until i know the specifics of what you believe Mendalian genetics erects as a barrier i have to stick with the things i know best.
That's fine, I am use to anecdotal evidence being passed for a demonstrated mechanism.
i think that sufficiently proves my contention until i learn more about exactly what it is about Mendelian genetics that you think a barrier between classes.
....
You might try this:
Human Genome Project Information
At the top of the page are the human chromosomes, if you want to know what happens when one of them has a genetic mutation just click on the number. If you like they will send you a poster with all of the diseases and disorders associated with they various chromosomes. Here are the ones associated with Human Chromosome 21 believed to be the one containing the genes that make us unique.
Human Chromosome 21
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