Give me one beneficial mutation.

3sigma

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Heaven-Sent

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If that doesn’t satisfy you, you could read through some of these other 10,000 odd papers. Did you think that there wasn't any evidence? Did you even think to look?

I spent 30 minutes reading that article, and I'm still stumped where the beneficial mutation is. Maybe I missed it.

the nylon eating bacteria comes to mind.

My cat eats cat food, but will also eat yogurt if I drop it on the floor. I made a smoothie earlier and some split on the ground, she ate that too. Like a vacuum cleaner, but no work involved.
 
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3sigma

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I spent 30 minutes reading that article, and I'm still stumped where the beneficial mutation is. Maybe I missed it.
So you are claiming you began reading the paper 10 minutes before I even posted a link to it? That’s impressive prescience. Maybe you should contact JREF.

I can’t help you with understanding the paper. Perhaps you should take some university courses in biology.
 
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Heaven-Sent

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So you are claiming you began reading the paper 10 minutes before I even posted a link to it? That’s impressive prescience. Maybe you should contact JREF.

I can’t help you with understanding the paper. Perhaps you should take some university courses in biology.

I asked what the beneficial mutation was and you cannot give me an answer.
 
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sfs

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Off the top of my head, beneficial mutations in humans:

Mutations in a regulatory region for the gene that codes for lactase. These mutations have occurred and been selected for in several different cattle-herding populations around the world, including northern Europeans. They have the effect of making the people who have them lactose-tolerant as adults (which is not the original state for humans).

Mutations in at least a dozen different genes controlling skin pigmentation, which have had the effect of tuning skin color by latitude.

Duplication of a gene for an amylase enzyme in saliva, that helps break down starchy foods. Populations with a history of a high-starch diet have acquired (on average) more copies of the gene.

A mutation in the gene EDAR in East Asians. No one has a clue why the mutation was beneficial, but it clearly was; the mutation probably contributes to the common East Asian hair texture, although there's no reason to think that fact has anything to do with why it was beneficial.
 
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3sigma

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I asked what the beneficial mutation was and you cannot give me an answer.
No, because I’m not a biologist. I suggest you ask a biologist or take some courses in biology if you want to understand why you can’t see the answer.

However, this excerpt appears to begin to address your question.

We found a beneficial mutation in the BoxG1 protein-binding site located near the P1 promoter of the glmUS operon. The mutation was a 1-bp insertion of an adenine in a tract of seven existing adenines. The mutation spread in its source population between generation 500, when it was rare or absent, and generation 1500, when it was present in almost every cell. The mutation persisted through 20,000 generations. It conferred a substantial fitness advantage, on the order of 5%, when transduced into the ancestor.
Again, don’t come to me looking for answers. Read the other papers, ask biologists, take university courses and learn enough biology to see why there is evidence of beneficial mutations occurring.
 
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sbvera13

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Why be satisfied with only one? Oh, and just for kicks, this list includes mutations in humans only.

apoaA-I : Prevents HDL Cholesterol related illnesses.

CCR5-delta 32
: Provides near immunity to AIDS.

Tetrachromacy
: Allows women with this mutation to see normally invisible wavelengths of light.

LRP5(V171) : Creates extremely dense, strong bones and immunity to osteoporosis.

EGLN1 and PPARA : Allows the Tibetans who carry this mutation to live at extreme high altitudes without suffering from altitude sickness.
 
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sbvera13

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I spent 30 minutes reading that article, and I'm still stumped where the beneficial mutation is. Maybe I missed it.
In a nutshell: e-coli bacteria were kept in jars for a very, very long time., and only given 2 kinds of food. A teeny, tiny bit of Glucose, which they eat very well, and lots and lots of Cellulose, which they can barely eat at all. After about 20 years, the bacteria mutated to be able to eat the glucose and cellulose equally well. This is very beneficial for them, because now they have lots of yummy food.

ps. Oh, and it was done in laboratory conditions, with genetic testing and meticulous record keeping. They mutated, scientists know what gene did it, when it did it, why it happened, and how it works.
 
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Heaven-Sent

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Off the top of my head, beneficial mutations in humans:

Mutations in a regulatory region for the gene that codes for lactase. These mutations have occurred and been selected for in several different cattle-herding populations around the world, including northern Europeans. They have the effect of making the people who have them lactose-tolerant as adults (which is not the original state for humans).

Mutations in at least a dozen different genes controlling skin pigmentation, which have had the effect of tuning skin color by latitude.

Duplication of a gene for an amylase enzyme in saliva, that helps break down starchy foods. Populations with a history of a high-starch diet have acquired (on average) more copies of the gene.

A mutation in the gene EDAR in East Asians. No one has a clue why the mutation was beneficial, but it clearly was; the mutation probably contributes to the common East Asian hair texture, although there's no reason to think that fact has anything to do with why it was beneficial.

All of these are just using added existing code.
 
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Heaven-Sent

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No, because I’m not a biologist. I suggest you ask a biologist or take some courses in biology if you want to understand why you can’t see the answer.

However, this excerpt appears to begin to address your question.


Again, don’t come to me looking for answers. Read the other papers, ask biologists, take university courses and learn enough biology to see why there is evidence of beneficial mutations occurring.

lol? So you just googled beneficial mutation and threw me a link or what? You don't even know what the article is about, and you want me to figure it out? I would rather debate with someone who knows what they're talking about. I am not an expert in the field of biology, but I am willing to take one on.
 
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sfs

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All of these are just using added existing code.
No, if they just used existing code they wouldn't be mutations. A mutation means that the new code is different from the old code. Are you just making up replies here?

Look, you asked for a single beneficial mutation. You have been given many. Try dealing with that fact.
 
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sbvera13

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All of these are just using added existing code.
SWOOSH go the goalposts as they move across the field!

You asked for mutations. You got them. You did not ask for spontaneously generated, unique protein codes.

BTW, the apoaA-I mutation DOES code for a new protein. Not sure if the code for it is novel or not though. I'm nto enough of an expert to say.
 
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Heaven-Sent

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In a nutshell: e-coli bacteria were kept in jars for a very, very long time., and only given 2 kinds of food. A teeny, tiny bit of Glucose, which they eat very well, and lots and lots of Cellulose, which they can barely eat at all. After about 20 years, the bacteria mutated to be able to eat the glucose and cellulose equally well. This is very beneficial for them, because now they have lots of yummy food.

ps. Oh, and it was done in laboratory conditions, with genetic testing and meticulous record keeping. They mutated, scientists know what gene did it, when it did it, why it happened, and how it works.

Ah, thank you for breaking it down. So the expirement was a bacteria, that could eat both glucose and cellulose, that was put in a jar to see if it could eat glucose and cellulose? Who funds these projects?

Btw, I will research your previous post and get back to you on that one.
 
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