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and not every chemoreceptor is able to detect an odor molecule too:
"that can activate a nearby nerve fiber if the chemosensor is a specialized sensory receptor cell, such as the taste receptor in a taste bud[2][3] or in an internal peripheral chemoreceptor such as the carotid body"
Chemoreceptor - Wikipedia
as i said: an olfactory receptor is a special kind of receptor and not every receptor is able to bind an odor molecule.
and yet: we will need a new mutation in moving to olfactory receptor from non olfactory receptor.
again: if the first mutation is neutral (and i showed here why it need to be neutral), then we need to calculate what is the chance to get about 3 different parts by a nautral evolution. and the chance is very low.New mutations happen all the time, so again, relevance?
You sincerely don't demonstrate that. A mutation that results in a new structure that has no function isn't neutral. Neutral mutations, by definition, have no recognizable impact on physiology.again: if the first mutation is neutral (and i showed here why it need to be neutral)
-_- the first mutation being neutral would be irrelevant; chances are it wouldn't even alter the impact of the next benign or detrimental mutation on that gene very much. That is, unless you confuse the generation of a useless structure with neutrality., then we need to calculate what is the chance to get about 3 different parts by a nautral evolution. and the chance is very low.
Show your work.again: if the first mutation is neutral (and i showed here why it need to be neutral), then we need to calculate what is the chance to get about 3 different parts by a nautral evolution. and the chance is very low.
Amazing.
How were all the different chemoreceptors Created, and what is your evidence?
You sincerely don't demonstrate that. A mutation that results in a new structure that has no function isn't neutral. Neutral mutations, by definition, have no recognizable impact on physiology.
Is it just me, or has your grammar and sentence structure gotten significantly worse? I'm actually starting to have trouble reading your posts, and that has not always been the case.i find no problem to consider it as a neutral mutation since it have no biological rule.
No, you don't get to redefine these established terms for the sake of discussion. I don't even understand why you want to, considering that the mutations you are describing would be considered mildly detrimental. Wouldn't it be more in your favor to argue that detrimental mutations would be a necessary part of the development of a benign structure, and thus natural selection couldn't possibly select for it? Why am I a better creationist than you, come on, a challenge would be nice.so for the sake of discussion lets consider any such mutation as neutral.
Also, you STILL have not demonstrated that the situation you are talking about has to happen for new structures to form. There is no demonstrable reason that a structure couldn't have various functions as it changes from start to "finish" (air quotes because evolution is a continuous process that never stops).
Consider a simple organism that uses surface cells with flagellae for orientation and locomotion. Suppose now that a surface cell becomes sensitized to some class of external chemicals by a mutation, and this sensitivity results in a cellular signal (biochemical transmitter, membrane depolarization, or whatever) being triggered when this class of chemicals is encountered. If this signal influences the surrounding flagellae, either to reduce or increase their motility, the organism will correspondingly move towards or away from, respectively, the sensitizing chemical signal.but we already seen that an olfactory receptor dont have any function by itself. so if we will consider it as step 1 (out of 3), then the first mutation (olfactory receptor) will be non functional.
We know that this structure is CURRENTLY dependent upon others to function, but you have yet to demonstrate that it isn't derived from a structure that functioned independently, or that it didn't develop until after the basic accessory structures it utilizes already existed.but we already seen that an olfactory receptor dont have any function by itself.
If we did that, we'd already be making a huge mistake.so if we will consider it as step 1 (out of 3),
You are ASSUMING that it started out this way, which is hugely erroneous. That's like saying that the first limb was a complete human arm missing an elbow.then the first mutation (olfactory receptor) will be non functional.
but we already seen that an olfactory receptor dont have any function by itself.
so if we will consider it as step 1 (out of 3), then the first mutation (olfactory receptor) will be non functional.
Suppose now that a surface cell becomes sensitized to some class of external chemicals by a mutation, and this sensitivity results in a cellular signal (biochemical transmitter, membrane depolarization, or whatever) being triggered when this class of chemicals is encountered.
We know that this structure is CURRENTLY dependent upon others to function, but you have yet to demonstrate that it isn't derived from a structure that functioned independently, or that it didn't develop until after the basic accessory structures it utilizes already existed.
Because cars only "evolve" in a metaphorical sense, not through the random variation of heritable traits followed by natural selection.again: we can compare it with a car for instance. if you agree that a car cant evolve stepwise then why we cant conclude the same for living systems?
What is design and how do you detect it in an object?so lets say that we will have a self replicating matter. do you think that it can evolve stepwise into a car?
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