What's wrong with half placentas again? To my knowledge, a large number of live-bearing vertebrates have placenta-like organs. Eutherian mammals just took them a few steps further than most.
Clue: "=/=" means DOES NOT EQUAL. This is the same thing I've been saying all along. Seems you just misunderstood it.
I'll take that as a compliment

As it happens, I do, in fact, have an idea what "similar" means. Most of my undergrad project was about finding similarities in sequences. (Granted, in protein sequences, not DNA. Then again, it's not
my experience that's relevant to ERV discussion, seeing as I didn't write those papers.)
Um... how does the fact that only a few chimp (or human) ERVs possess all the components of a retrovirus contradict evolution?
(And look how you bolded "underestimate". What were you trying to say with that?)
Which everyone has been saying all along...
Lolwut?
Psychobabble? You do know that the prefix "psycho-" is generally used in connection with, you know,
minds, not molecular genetics. Do you mean
technobabble? Either way, your lack of understanding doesn't make it "babble".
Here, have a retrovirus or two:
These are genome structures of an active, functional retrovirus, namely the two main varieties of human immunodeficiency virus (image from
Dan Stowell's hypertextbook on HIV).
These 2 genome structures are the result of erraneous algorithms, that use assumptions and are erraneous eg...Similar indels produced by horizontal insertion. There is plenty of research I can quote that shows many erraneous assumptions your algorithms are based on.
The whole thing is
thousands of base pairs. (In this case, around 10 thousand). There is plenty of diagnostic stuff in a retroviral genome, even if you strip away some of its components. The LTRs are
several hundred bp each, and as far as I can tell they are not called "repeats" because they are made of repetitive DNA, but because there is one at each end of a complete retrovirus. So a "solitary LTR" can still be quite recognisable. (You know, like you can probably still recognise your mother if you only see half her face.)
Again..a solitary LTR may be recognizable but being a remnant of a bigone engogenised virus is an assumption backed by erraneous algorithms and supported by more erraneous algorithms.
I don't really understand why it's a problem that most LTR retrothingies lack viral genes...
Don't worry, your researchers do not understand most stuff. They guess alot though!
What are your
specific problems with their
specific algorithms? It would be even better if you showed us an example. You know, the stuff commonly called "evidence".
I have, but you ignore it and request it over and over. That's why I had a few days off here. It is truly a waste of time talking science to an evolutionist.
I'll have another go so that you can ignore it and carry on as if evolutionists belong in biology labs...
However, the inference that shared intron positions reflect evolutionary conservation is complicated by the fact that intron insertion does not seem to be a strictly random process. A simple form of such non-randomness was taken into account in the parsimonious reconstruction of intron evolution by Monte Carlo simulation with intron insertion allowed only in a fraction of a gene's sequence (e.g. 10%). The results suggested a relatively small contribution of independent insertion in the same position in different lineage (parallel gain) to intron evolution (4,5).
Nevertheless, a greater role of parallel gain due to non-random intron insertion cannot be ruled out. The short sequence stretches flanking introns have significantly biased nucleotide frequencies which led to the notion of protosplice sites, the target sites for intron insertion (6,7). However, it remained unclear whether the consensus nucleotides flanking the splice junctions were remnants of the original protosplice sites or evolved convergently after intron insertion.
Conservation versus parallel gains in intron evolution
It is not about whom is right or wrong or is the latest right. The point is.. it is all no more credible than flavour of the month. This is another point you are unable to assimilate.
Non-plausible and ridiculous as in observed all the time? You talk like we've never seen a retrovirus infect a species more than once. Come on, how many HIV positive people live on this planet right now?
If you'd ever bothered to learn how sequence comparison is done, you'd be ashamed of yourself. Eggs and coconuts are
not labelled "similar" jut 'cause someone wants them to be so.
Yeah well your researchers use 40% & 60% similar as represented in algorithms as 'the same'. Now you say this is rubbish to the forum
just so I can show you evidence that I am correct.
Here is a little taste...
Specifically, 3,644 pairs (95.2%) among 3,828 possible pairs of these 88 sequences have <25% sequence identity. Within our dataset, the group with the highest sequence identity is the Mt plasmid group (mean 61.2% identity ± 41.8% s.d.), and the group with the smallest sequence identity is the telomerase group (mean 17.9%)
Phylogenetic profiles reveal evolutionary relationships within the âtwilight zoneâ of sequence similarity
Eggs and coconuts was an excellent comparison of what evolutionists call 'the same', actually...
There was an experiment I did recently, mostly for my own amusement. I took information on amino acid frequencies in real proteins (from
here, if you are interested), randomly generated a hundred protein sequences using those frequencies, and ran them all through BLASTp just to see what I'd get. These sequences, being random, were truly and honestly unrelated to anything in the database.
I got... pretty much nothing. None of them scored a match I would call convincing. When I did a conserved domain search on them, only 3 out of 100 matched anything at all, all 3 were only partial matches, and poor partial matches at that. Point being:
completely unrelated sequences of a decent length (mine were 300 amino acids)
are not bloody likely to deceive us into thinking they are related. If something looks like a piece of retrovirus, there's a good chance that that's exactly what it is.
Retroviruses don't insert in the real world?
News for you: the idea of common descent was founded almost entirely on functional traits. Having a function does not preclude being evidence for common ancestry. Function doesn't have to require so much similarity that homology and convergence become indistinguishable. (Cases in point: analogous enzymes, wing anatomies of flying vertebrates...)
Absolutly
How about God saw some old virus and thought, "hey, that could work..."

?
(P.S.: I
asked you not to write your responses inside a quote. You may have noticed that CF doesn't allow multilevel quoting, so if I wanted to use those paragraphs, I'd have to individually copy-paste them from your post. Sorry, but you are not worth that much effort. If you want me to read and reply to something, put it in a quotable place.)
My multi qoute is broke or something