A strange dichotomy, considering that processes using random variation and selection have a wide application in industrial design and manufacturing.
But then it's designed to do so, and only for details.
And industrial design and manufacturing is childs play compared to the product we call creation.
So the dichotomy is not strange at all, it is obvious.
Your comparison is weak.
And here we have some statements that reveal your complete ignorance of how the real theory of evolution is supposed to work.
Your following explanation boils down to the same thing.
Here is an answer I gave to a creationist who wanted me to explain how speciation occurs according to the real theory of evolution. He ignored it, of course, because it wasn't what he wanted to hear. Why don't you take a crack at it?
First, evolutionary biologists don't see speciation as a big deal.
Of course they do.
They assume it started from 1 kind / species of organism.
It seems to me you don't realise how utterly specialized the various organisms are.
Explaining this variety is obviously THE challenge for naturalists / evolutionnists.
Species aren't pre-existing categories into which creatures evolve; they are descriptive, not prescriptive.
Of course, but then they assume evolution.
When part of a population is subjected to different selection criteria than the rest, it will evolve to meet those new criteria.
Only if there's change.
No change to select is no change.
This change is supposed to have been caused by random mutations.
Yes, they can survive, they do survive, but they can not write specialized data by corrupting the existing data.
However, this is the belief and fundament of the assumed change (like new organs).
Selection doesn't change anything, changes (mutations) change things.
"Evolving to meet" suggests a direction, but there's no direction in random mutations.
If it evolves enough that the two groups are no longer interfertile, a new species is said to have formed.
Still, "Kind begets kind".
Where's the evidence that suggests otherwise?
Why believe otherwise?
Do you think it's progress when a kind looses interfertility?
But the process is slow and partial interfertility will last a long time. There is no "hard line" between species. That is why the determination of species is sometimes difficult and can be controversial.
Alright, I see what you mean.
Indeed, kinds or species, where does taxonomy draw the lines?
How does it happen? No offspring is exactly the same as its parent. In each generation the population will present a range of variants to the environment for selection. For a given heritable trait, the length of a limb, say, the distribution of variation will be random (think "bell curve"). Most variation will be near the average, with outriders at either extreme, like a bell curve. If selection criteria are stable, the central group will survive to reproduce, the outriders not so much. However, if the selection criteria change, there will be already in the population at least a few individuals on the edges to take advantage of the situation. As they reproduce more successfully, generation by generation, than the individuals nearer the original central tendency and on the other side of it, the central tendency will shift in that direction, producing more outriders to take advantage of further shifts in selection criteria.
Fine, but this is no explanation for different organs in different kinds / species.
You still have that to explain "without God doing it".
You know birds for example have all kinds of specialized organs to be a bird.
How did the data for a fertilized egg cell growing into a bird come about?
Why would assumed systems in development (i.e. not yet operative) dominate the gene pool of the assumed "not yet bird"?
These are the big questions obviously.
And where is the compelling evidence for it?
And so on. How long? Hard to say.
Obviously the naturalistic model need a LOT of time, like millions, even billions of years.
That's why they torture the data as long as it takes to confess to it.
It depends on the size and diversity of the gene pool and the degree of selection pressure.
Selection pressure can be too high too, and species go extinct.
Maintaining the diversity of the gene pool is key to to successful evolution; a population of clones would generate little or no variation and would not be able to evolve.
Hence you need mutations for the actual change.
Since these mutations are random / have no direction, it is not realistic to expect them to come up with genius systems.
Let alone in the abundance we find in living nature.
Claiming otherwise needs a proper explanation and evidence to support it.
Gene pool diversity is also reduced by the normal action of natural selection. That's where mutations and other contributors of diversity come in.
I want to thank you for trying to explain things.
It is however no explanation of what we find in living nature.
The obvious problem is accounting for species / kind specific traits like organs and incorporated systems.
And why choose this model then over design and manufacturing?
Look, i know i'm not an expert and i know i'm probably annoying.
But when there's no explanation for speciation (ranging from moss to bird, from feline to arachnid, all of them including us) i.e. the writing of DNA, let alone the arise of the system of DNA replication itself, then why believe it?
Anyway, we'd better stop this, i don't think it's useful to continue.
God bless.