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Does science actually admit "design"?

OldWiseGuy

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I don't have to know how creation works.
 
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OldWiseGuy

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I have another theory (the layman's definition). What if those "cell-to-cell" nerve signals generated by the heart spike during a sudden shocking event and pass to the left RLN via the aortic arch then to the larynx? Remember I'm just brainstorming here as my intuition tells me the design has purpose, just as my intuition told me that the eye had to be dissected for the 'blind spot' to be realized.
 
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VirOptimus

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That not a theory under any definition, thats a wild specualtion with no basis in reality.
 
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OldWiseGuy

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That not a theory under any definition, thats a wild specualtion with no basis in reality.

Here's the point. The entire body has purposeful design, therefore the position of the left RLN is right where it's supposed to be regardless of the opinions of evolutionists. If it's position under the aortic arch was necessary during early growth and it was pulled down subsequently to it's final position then it is entirely appropriate to be where it is. As has been noted by others it cannot somehow disconnect and then reconnect later to suit the evolutionists. That said I believe it has a function that requires it to be right where it is. We just don't know what that function is exactly. What is clear is that it makes connections along it's length to various organs. What is needed of course is for the evolutionists to disconnect it and reconnect it to their liking and then observe the results.
 
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PsychoSarah

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Here's the point. The entire body has purposeful design, therefore the position of the left RLN is right where it's supposed to be regardless of the opinions of evolutionists.
One of the biggest signs, in my opinion, that a structure is either vestigial or doesn't need to adhere to strict structural guidelines for its function is variation. A lot of the papers I find that mention the left RLN mention how variations in it make heart surgery more difficult for some patients than others.

To be blunt, though, there are much better examples of flaws in anatomy of humans and other animals that I view focusing on this one is pointless. For example, the palmaris longis muscle is absent in over 10% of humans, and it is not uncommon for it to be absent in one arm and present in the other. The muscle is tiny and doesn't seem to impact physical strength much, if at all.

-_- or how about the fact that the pathway to the stomach and the lungs shares a tube part of the way? It would have been trivial to design organisms with separate pathways for breathing and eating, and the detriment of that shared pathway should be obvious to any designer worth their salt.

Our feet have too many unnecessary bones, which results in the arches of our feet breaking down more easily than, say, ostrich feet do.

-_- you do know that it is trivial to determine where nerves innervate, right? Furthermore, that its position is a consequence of embryonic development doesn't suddenly make it the most efficient and effective position for the nerve to be in. It just explains why it ends up that way. You are acting as if the designer you believe in didn't design the process of embryonic development at all.
 
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tas8831

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I don't have to know how creation works.


OK. I don't have to know how evolution works.

Now we are on a level playing field, and the only thing we can now do is present our evidence.

Here is but a taste of mine:



I forget now who originally posted these on this forum, but I keep it in my archives because it offers a nice 'linear' progression of testing a methodology and then applying it:

The tested methodology:

Science 25 October 1991:
Vol. 254. no. 5031, pp. 554 - 558

Gene trees and the origins of inbred strains of mice

WR Atchley and WM Fitch

Extensive data on genetic divergence among 24 inbred strains of mice provide an opportunity to examine the concordance of gene trees and species trees, especially whether structured subsamples of loci give congruent estimates of phylogenetic relationships. Phylogenetic analyses of 144 separate loci reproduce almost exactly the known genealogical relationships among these 24 strains. Partitioning these loci into structured subsets representing loci coding for proteins, the immune system and endogenous viruses give incongruent phylogenetic results. The gene tree based on protein loci provides an accurate picture of the genealogical relationships among strains; however, gene trees based upon immune and viral data show significant deviations from known genealogical affinities.

======================

Science, Vol 255, Issue 5044, 589-592

Experimental phylogenetics: generation of a known phylogeny

DM Hillis, JJ Bull, ME White, MR Badgett, and IJ Molineux
Department of Zoology, University of Texas, Austin 78712.

Although methods of phylogenetic estimation are used routinely in comparative biology, direct tests of these methods are hampered by the lack of known phylogenies. Here a system based on serial propagation of bacteriophage T7 in the presence of a mutagen was used to create the first completely known phylogeny. Restriction-site maps of the terminal lineages were used to infer the evolutionary history of the experimental lines for comparison to the known history and actual ancestors. The five methods used to reconstruct branching pattern all predicted the correct topology but varied in their predictions of branch lengths; one method also predicts ancestral restriction maps and was found to be greater than 98 percent accurate.

==================================

Science, Vol 264, Issue 5159, 671-677

Application and accuracy of molecular phylogenies

DM Hillis, JP Huelsenbeck, and CW Cunningham
Department of Zoology, University of Texas, Austin 78712.

Molecular investigations of evolutionary history are being used to study subjects as diverse as the epidemiology of acquired immune deficiency syndrome and the origin of life. These studies depend on accurate estimates of phylogeny. The performance of methods of phylogenetic analysis can be assessed by numerical simulation studies and by the experimental evolution of organisms in controlled laboratory situations. Both kinds of assessment indicate that existing methods are effective at estimating phylogenies over a wide range of evolutionary conditions, especially if information about substitution bias is used to provide differential weightings for character transformations.



We can ASSUME that the results of an application of those methods have merit.


Application of the tested methodology:

Implications of natural selection in shaping 99.4% nonsynonymous DNA identity between humans and chimpanzees: Enlarging genus Homo

"Here we compare ≈90 kb of coding DNA nucleotide sequence from 97 human genes to their sequenced chimpanzee counterparts and to available sequenced gorilla, orangutan, and Old World monkey counterparts, and, on a more limited basis, to mouse. The nonsynonymous changes (functionally important), like synonymous changes (functionally much less important), show chimpanzees and humans to be most closely related, sharing 99.4% identity at nonsynonymous sites and 98.4% at synonymous sites. "



Mitochondrial Insertions into Primate Nuclear Genomes Suggest the Use of numts as a Tool for Phylogeny

"Moreover, numts identified in gorilla Supercontigs were used to test the human–chimp–gorilla trichotomy, yielding a high level of support for the sister relationship of human and chimpanzee."



A Molecular Phylogeny of Living Primates

"Once contentiously debated, the closest human relative of chimpanzee (Pan) within subfamily Homininae (Gorilla, Pan, Homo) is now generally undisputed. The branch forming the Homo andPanlineage apart from Gorilla is relatively short (node 73, 27 steps MP, 0 indels) compared with that of thePan genus (node 72, 91 steps MP, 2 indels) and suggests rapid speciation into the 3 genera occurred early in Homininae evolution. Based on 54 gene regions, Homo-Pan genetic distance range from 6.92 to 7.90×10−3 substitutions/site (P. paniscus and P. troglodytes, respectively), which is less than previous estimates based on large scale sequencing of specific regions such as chromosome 7[50]. "




Catarrhine phylogeny: noncoding DNA evidence for a diphyletic origin of the mangabeys and for a human-chimpanzee clade.

"The Superfamily Hominoidea for apes and humans is reduced to family Hominidae within Superfamily Cercopithecoidea, with all living hominids placed in subfamily Homininae; and (4) chimpanzees and humans are members of a single genus, Homo, with common and bonobo chimpanzees placed in subgenus H. (Pan) and humans placed in subgenus H. (Homo). It may be noted that humans and chimpanzees are more than 98.3% identical in their typical nuclear noncoding DNA and probably more than 99.5% identical in the active coding nucleotide sequences of their functional nuclear genes (Goodman et al., 1989, 1990). In mammals such high genetic correspondence is commonly found between sibling species below the generic level but not between species in different genera."​


Your turn.


And please don't reply with something stupid like 'are we related to mice?' or 'the mice are still mice' or some other statement of your ignorance of the subject coupled with a desire to rescue creationism - just present your positive, supporting evidence.

Like I did.
 
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tas8831

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The plagiarist rants!
 
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tas8831

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Here's the point. The entire body has purposeful design, therefore the position of the left RLN is right where it's supposed to be regardless of the opinions of evolutionists.

LOL!

My gosh - a 135 IQ and this guy RELIES on question begging!


Still waiting:

" This is a visceral reaction (the 'mind' of the body) influencing the function of the throat and voice box without the direction of the brain. The signal gets there via the RLN in the case of the giraffe."

Please provide evidence that 1. such a neural pathway exists and 2. that is actually functions in the manner you keep asserting.
 
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FrumiousBandersnatch

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You apparently missed the point of the OP, which was not about design in science, but about purposeful design in nature (evolution).

I say 'apparently' because (charitably) it's hard to believe you really didn't know what you were doing. What a waste of time...

Frankly, the OP is just an attempt to suggest that the casual teleological or teleonomic mode of speech (Dennett's 'Intentional Stance') should be taken literally.
 
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OldWiseGuy

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If man and apes are so closely 'related' we should be able to tweak the genome of apes and produce humans from their species, and vice versa.
 
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FrumiousBandersnatch

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Didn't we copy the design of bird wings so that airplanes can fly? We deem our wings designed, but bird wings aren't? I think it would be proper to say that we copied the functional design of bird wings.
I'm happy with that wording (although the wings of the first aircraft owed little or nothing to bird wings) - I have no problem with evolutionary 'design'; it's a term in common use outside biology.

The reason it's generally avoided by evolutionists talking to creationists is that creationists insist that the use of 'design' implies an intelligent designer - and as we know, evolutionary processes are not intelligent.
 
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OldWiseGuy

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Provide evidence that it doesn't.
 
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tas8831

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Provide evidence that it doesn't.

Already did and you ignored it. As usual, since you are not an honest discussant.



Repetition of debunked assertions
Responding only to a small or even tangential issue in very long posts and ignoring the rest
Asking others for sources then ignoring the sources when they are given
Refusing to provide actual support for claims
Playing martyr when people expose and exploit your trollish habits
etc.


Like this:

The topic is sounds, not Speech and Language.

Was the entirety of the IQ of 135 creationist super-genius' response to this:



You implied you know about Gray's anatomy, right?

By the way - I ALREADY provided you with a source, but you probably just didn't bother to read it. Creationists are like that - they don't actually want to know how little they know.


So, since you think Google U makes you the expert you pretend to be, I found these in a couple of minutes:

The Neural Basis of Speech and Language (this is the one I linked for you before and you clearly ignored or more likely could not understand)
http://samples.jbpub.com/9781449652678/74738_CH02_FINAL.pdf


Vagus Nerve
http://www.caam.rice.edu/~cox/wrap/vagusnerve.pdf

Why, even Wiki:
General visceral afferent fibers - Wikipedia


From here:


General visceral afferent fibers


The general visceral afferent fibers (GVA) conduct sensory impulses (usually pain or reflex sensations) from the internal organs, glands, and blood vessels to the central nervous system.[1] They are considered to be part of the autonomic nervous system. However, unlike the efferent fibers of the autonomic nervous system, the afferent fibers are not classified as either sympathetic or parasympathetic.[2]

GVA fibers create referred pain by activating general somatic afferent fibers where the two meet in the posterior grey column.

The cranial nerves that contain GVA fibers include the facial nerve (CN VII), the glossopharyngeal nerve (CN IX), and the vagus nerve (CN X).[3]

Generally, they are insensitive to cutting, crushing or burning, excessive tension in smooth muscle and some pathological conditions produce visceral pain (referred pain).[4]

Pathway
Abdomen

In the abdomen, general visceral afferent fibers usually accompany sympathetic efferent fibers. This means that a signal traveling in an afferent fiber will begin at sensory receptors in the afferent fiber's target organ, travel up to the ganglion where the sympathetic efferent fiber synapses, continue back along a splanchnic nerve from the ganglion into the sympathetic trunk, move into a ventral ramus via a white ramus communicans, and finally move into the mixed spinal nerve between the division of the rami and the division of the roots of the spinal nerve. The GVA pathway then diverges from the sympathetic efferent pathway, which follows the ventral root into the spinal column, by following the dorsal root into the dorsal root ganglion, where the cell body of the visceral afferent nerve is located.[5] Finally, the signal continues along the dorsal root from the dorsal root ganglion to a region of gray matter in the dorsal horn of the spinal column where it is transmitted via a synapse to a neuron in the central nervous system.[2]

The only GVA nerves in the abdomen that do not follow the above pathway are those that innervate structures in the distal half of the sigmoid colon and the rectum. These afferent fibers, instead, follow the path of parasympathetic efferent fibers back to the vertebral column, where the afferent fibers enter the S2-S4 sensory (dorsal root) ganglia followed by the spinal cord.[5]
Pelvis

The course of GVA fibers from organs in the pelvis, in general, depends on the organ's position relative to the pelvic pain line. An organ, or part of an organ, in the pelvis is said to be "above the pelvic pain line" if it is in contact with the peritoneum, except in the case of the large intestine, where the pelvic pain line is said to be located in the middle of the sigmoid colon.[6] GVA fibers from structures above the pain line follow the course of the sympathetic efferent fibers, and GVA fibers from structures below the pain line follow the course of the parasympathetic efferents.[6] Pain from the latter fibers is less likely to be consciously experienced.[6]


References

Moore, Keith; Anne Agur (2007). Essential Clinical Anatomy, Third Edition. Lippincott Williams & Wilkins. p. 635. ISBN 0-7817-6274-X.
Moore, Keith; Anne Agur (2007). Essential Clinical Anatomy, Third Edition. Lippincott Williams & Wilkins. pp. 34–35. ISBN 0-7817-6274-X.
Mehta, Samir et al. Step-Up: A High-Yield, Systems-Based Review for the USMLE Step 1. Baltimore, MD: LWW, 2003.
Susan,, Standring,. Gray's anatomy : the anatomical basis of clinical practice. ISBN 9780702052309. OCLC 920806541.
Moore, K.L., & Agur, A.M. (2007). Essential Clinical Anatomy: Third Edition. Baltimore: Lippincott Williams & Wilkins. 180. ISBN 978-0-7817-6274-8
Moore, Keith; Anne Agur (2007). Essential Clinical Anatomy, Third Edition. Lippincott Williams & Wilkins. p. 220. ISBN 0-7817-6274-X.​


Same source, on the special visceral afferent fibers - uh uh! this one actually mentions the larynx! Maybe this will be my Waterloo, and will provide evidence for the creationist's anatomical assertions?

Special visceral afferent fibers (SVA) are the afferent fibers that develop in association with the gastrointestinal tract.[1] They carry the special senses of smell (olfaction) and taste (gustation). The cranial nerves containing SVA fibers are the olfactory nerve (I), the facial nerve (VII), the glossopharyngeal nerve (IX), trigeminal nerve (V) and the vagus nerve (X). The facial nerve receives taste from the anterior two-thirds of the tongue; the glossopharyngeal from the posterior third. SVA fibers in the vagus originate in the larynx and pharynx.[2] The sensory processes, using their primary cell bodies from the inferior ganglion, send projections to the medulla, from which they travel in the tractus solitarius, later terminating at the rostral nucleus solitarius.[3]​


Nope. Just more evidence that the creationist is out of his depth and that his claim of studying anatomy was a farce.

And wiki again on the RLN:

Recurrent laryngeal nerve - Wikipedia


Now please provide an actual source that shows that motor impulses for vocalizations can be produced anywhere other than the Nucleus ambiguus (which in turn receives inputs from the motor speech area).

Surely you know what that is, what with your keen grasp of the relevant anatomy, right?


Of course, you would have had to understand anatomy enough to know what to search for (e.g., vagus nerve, visceral afferents, etc.) which you obviously do not (and remember that according to you, if something is obvious it must be so). This is why your keyword search technique has, every time I have seen you employ it thus far, ended up making you look foolish for linking to articles that actually undermine your position.


Funny - note that I was easily able to provide sources that actually do support my position, yet the creationist cannot seem to be able to do it ever.


PREDICTION - this will be responded to with first a one or two liner blow off, probably bringing up some ancillary subject, and perhaps later with a tangential link to a creationist essay.

Bets?​


Those are legitimate sources, reporting on decades/centuries of amassed knowledge of the nervous system.

You? You just splurt out 'brainstorming' based on wishful thinking/face-saving and under-girded by the Dunning-Kruger effect.



Still waiting:

" This is a visceral reaction (the 'mind' of the body) influencing the function of the throat and voice box without the direction of the brain. The signal gets there via the RLN in the case of the giraffe."

Please provide evidence that 1. such a neural pathway exists and 2. that is actually functions in the manner you keep asserting.


Your turn - and please, stop the beta-like dodging and burden shifting.

Or just admit that you've got nothing and scamper back to the Christian-only forums where you are an intellectual superstar.​
 
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tas8831

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If man and apes are so closely 'related' we should be able to tweak the genome of apes and produce humans from their species, and vice versa.


100% inability to actually address the evidence noted.

100% lack of evidence presented for your phony beliefs also noted.

If man was made from dust as you believe, then you should be able to show how it was done.

Now cometh the trolling.
 
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tas8831

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I have another theory (the layman's definition). What if those "cell-to-cell" nerve signals generated by the heart


The heart doesn't generate 'nerve signals.'


You lose.

Again.


Instead of 'brainstorming' from a position of total ignorance, how about you read some of the references I have linked/quoted for you for a change?


Are you afraid that if you actually learn some basic science that you will finally see how truly foolish and simple-minded your anti-evolution positions are?
 
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OldWiseGuy

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tas8831

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I see pshun24 is back. He is ignoring me because I do not hold back when I catch him doing something dishonest, as he has been doing for many years at multiple forums. Someone he is not ignoring ought to ask him why he misrepresented the Stern and Susman paper - I was not the only one to find his dishonesty and document it.
 
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