No there is conspiracy theories and kiddes filling out the VAERS (think I spelled that wrong, sorry) form, antecedental reports are not proof.
If by Kiddes you mean kiddies you are mistaken on that count and the conspiracy theory remark.
These are the writers of the excerpt below. Google them. They are extremely creditable
Michael Palmer MD, Sucharit Bhakdi MD, Stefan Hockertz PhD
This is where you will find the PDF in which the excerpt below is taken.
doctors4covidethics.org
Here is an excerpt. Below are their resources. Pay close attention to them. It is strongly suggested that you follow the link to the PDF and check out all the resources yourself.
3.1.3.1 Fatalities reported in connection with COVID vaccines. Within just five months of the onset of vaccinations, EudraVigilance has accumulated 12,886 deaths in connec16 tion with the COVID-19 vaccines, of which the Pfizer vaccine accounted for almost half (6,306). In the same time period, VAERS has run up 4,406 deaths in all; of these, 91% were associated with the mRNA vaccines, with Pfizer accounting for 44% and Moderna for 47% of the total. It is impossible to know what percentage of all fatalities that occur after vaccination will actually be reported to VAERS or EudraVigilance. However, note that the 4,406 COVID vaccine-related fatalities accrued by VAERS during just the past 5 months exceed the cumulative total of all other vaccines combined, over the entire previous 20 years. It is therefore clear that these vaccines are far and away the most deadly ones in history— quite predictably so, and all for a disease whose case fatality rate does not exceed that of influenza [1, 38].
3.1.3.2 Severe events related to disrupted blood clotting. The litany of diagnoses in both databases that indicate pathological activation of blood clotting is almost endless— heart attacks, strokes, thromboses in the brain and in other organs, pulmonary embolism; but also thrombocytopenia and bleeding, which result from excessive consumption of thrombocytes and of coagulation factors in disseminated intravascular coagulation. These disease mechanisms caused many of the fatalities summarized above; in other cases, they caused severe acute disease, which will in many cases leave behind severe disability.
3.1.3.4 Severe adverse reactions among adolescents. In the age group of 12-17 years, two deaths likely related to the Pfizer vaccine were already reported to EudraVigilance. Also in this age group, there were 16 cases of myocarditis, all in males, and 28 cases of seizures among both sexes, 3 of them reported as life-threatening. There also were a few cases of stroke, myocardial infarction, and severe inflammatory disease. While the numbers of adverse events are much lower than those among adults, this is simply due to the hitherto far lower rates of vaccination in this age group. Should systematic vaccination be green-lighted for adolescents, we must expect these numbers to rapidly climb to a level resembling that seen in adults.
3.1.3.5 Miscarriages. As of June 21st, 2021, EudraVigilance lists 325 cases of miscarriage among vaccinated pregnant women. While it is difficult to ascertain by just how much vaccination will raise the rate of miscarriage, most of these cases were reported by healthcare professionals, who evidently considered a connection to the vaccine at least plausible. This series of cases alone would be reason enough to pause the vaccinations and investigate3.2.4 Reproductive toxicity. Reproductive toxicity was assessed using only one species (rats) and on only small numbers of animals (21 litters). A greater than twofold increase in pre-implantation loss of embryos was noted, with a rate of 9.77% in the vaccine group, compared to 4.09% in the control group. Instead of merely stating [30, p. 50] that the higher value was “within historical control data range,” the study should have stated unambiguously whether or not this difference was statistically significant; and if it was not, the number of experiments should have been increased to ensure the required statistical power. The same applies to the observations of “very low incidence of gastroschisis, mouth/jaw malformations, right sided aortic arch, and cervical vertebrae abnormalities.” Overall, these studies are inadequately described and apparently were also inadequately carried out. 3.2.5 Autoimmunity. Exposure to the vaccine will lead to cell damage due to the cationic lipids, and also to the immune attack on cells producing the spike protein. From the cells undergoing destruction, proteins and other macromolecules will be released; such material must then be cleared away by macrophages. When the clearing system is overloaded because of excessive cell damage and apoptosis (cell death), then the accumulation of cellular debris will lead to chronically excessive type I interferon release; this, in turn, will trigger further inflammation. With time, some macromolecules in the debris will become targets for the formation of autoantibodies and the activation of autoreactive cytotoxic T cells—they will begin to function as auto-antigens. This then leads to further tissue damage and the release of more autoantigens—autoimmune disease will develop. Such an outcome is particularly likely in immunocompromised people or in those who are genetically predisposed to autoimmune disease (e.g. those with the HLA-B27 allele). The risk of autoimmunity induced by BNT162b2 could be adequately addressed only in long-term studies; as with fertility or cancer, the very short period of preclinical and clinical testing means that we are flying blind. It should go without saying that all of these risks are particularly grave with children, adolescents, and young adults.
Here is the closing statement of the paper and the resources references to which it was based. For those who are interested. The first paragraph is linked to the original PDF that was posted above in part. And a few of the references were also linked to their origin.
Overall, it is clear that the risk of ADE is recognized in theory but is not addressed in practice. Given the abundant evidence of ADE with experimental SARS vaccines, this is completely irresponsible.
References [1] J. P. A. Ioannidis: Infection fatality rate of COVID-19 inferred from seroprevalence data. Bull. World Health Organ. (2020), BLT.20.265892. url:
https://www.who.int/bulletin/ online_first/BLT.20.265892.pdf. [2] J. P. A. Ioannidis: Reconciling estimates of global spread and infection fatality rates of COVID-19: An overview of systematic evaluations. Eur. J.
Clin. Invest. 5 (2021), e133554. pmid: 33768536. [3] CDC COVID-19 Response Team: Coronavirus Disease 2019 in Children - United States, February 12-April 2, 2020. MMWR. Morbidity and mortality weekly report 69 (2020), 422– 426. pmid:
32271728. [4] S. Tsabouri et al.: Risk Factors for Severity in Children with Coronavirus Disease 2019: A Comprehensive Literature Review. Pediatric clinics of North America 68 (2021), 321–338. pmid:
33228941. 20 [5] J. Y. Abrams et al.: Multisystem Inflammatory Syndrome in Children Associated with Severe Acute Respiratory Syndrome Coronavirus 2: A Systematic Review. J. Pediatr. 226 (2020), 45– 54. pmid:
32768466. [6] P. A. McCullough et al.: Multifaceted highly targeted sequential multidrug treatment of early ambulatory high-risk SARS-CoV-2 infection (COVID-19). Reviews in cardiovascular medicine 21 (2020), 517–530. pmid: 33387997. [7] C. Bernigaud et al.: Oral ivermectin for a scabies outbreak in a long-term care facility: potential value in preventing COVID-19 and associated mortality. Br. J. Dermatol. 184 (2021), 1207–1209. pmid:
33454964. [8] Anonymous: WHO advises that ivermectin only be used to treat COVID-19 within clinical trials. 2021. url:
https://www.who.int/news-room/feature-stories/detail/whoadvises-that-ivermectin-only-be-used-to-treat-covid-19-within-clinicaltrials. [9] J. Flood et al.: Paediatric multisystem inflammatory syndrome temporally associated with SARS-CoV-2 (PIMS-TS): Prospective, national surveillance, United Kingdom and Ireland, 2020. The Lancet regional health. Europe 3 (2021), 100075. pmid: 34027512. [10] N. K. Shrestha et al.: Necessity of COVID-19 vaccination in previously infected individuals. medRxiv (2021). doi: 10.1101/2021.06.01.21258176. [11] S. S. Nielsen et al.: SARS-CoV-2 elicits robust adaptive immune responses regardless of disease severity. EBioMedicine 68 (2021), 103410. pmid: 34098342. [12] A. Grifoni et al.: Targets of T Cell Responses to SARS-CoV-2 Coronavirus in Humans with COVID-19 Disease and Unexposed Individuals. Cell 181 (2020), 1489–1501.e15. pmid: 32473127. [13] N. Le Bert et al.: SARS-CoV-2-specific T cell immunity in cases of COVID-19 and SARS, and uninfected controls. Nature 584 (2020), 457–462. pmid: 32668444. [14] S. Cao et al.: Post-lockdown SARS-CoV-2 nucleic acid screening in nearly ten million residents of Wuhan, China. Nat. Commun. 11 (2020), 5917. pmid: 33219229. [15] R. Wölfel et al.: Virological assessment of hospitalized patients with COVID-2019. Nature 581 (2020), 465–469. pmid: 32235945. [16] K. Basile et al.: Cell-based culture of SARS-CoV-2 informs infectivity and safe de-isolation assessments during COVID-19. Clin. Infect. Dis. (2020). pmid: 33098412. [17] Anonymous: Covid: Secret filming exposes contamination risk at test results lab. 2021. url:
Covid: Secret filming exposes contamination risk at test results lab. [18] K. G. Andersen et al.: The proximal origin of SARS-CoV-2. Nat. Med. 26 (2020), 450–452. doi: 10.1038/s41591-020-0820-9. [19] B. Sørensen et al.: Biovacc-19: A Candidate Vaccine for Covid-19 (SARS-CoV-2) Developed from Analysis of its General Method of Action for Infectivity. QRB Discovery 1 (2020). doi: 10.1017/qrd.2020.8. [20] B. Sørensen et al.: The evidence which suggests that this is no naturally evolved virus. Preprint (2020). url: https : / / www . minervanett . no / files / 2020 / 07 / 13 / TheEvidenceNoNaturalEvol.pdf. [21] L. Yan et al.: Unusual Features of the SARS-CoV-2 Genome Suggesting Sophisticated Laboratory Modification Rather Than Natural Evolution and Delineation of Its Probable Synthetic Route. Preprint (2020). doi: 10.5281/zenodo.4028829. 21 [22] L. Yan et al.: SARS-CoV-2 Is an Unrestricted Bioweapon: A Truth Revealed through Uncovering a Large-Scale, Organized Scientific Fraud. Preprint (2020). doi: 10.5281/zenodo. 4073131. [23] S. Yang and R. E. Rothman: PCR-based diagnostics for infectious diseases: uses, limitations, and future applications in acute-care settings. Lancet Infect. Dis. 4 (2004), 337–48. pmid: 15172342. [24] V. M. Corman et al.: Detection of 2019 novel coronavirus (2019-nCoV) by real-time RT-PCR. Euro Surveill. 25 (2020). pmid: 31992387. [25] Anonymous: Corman-Drosten review report. 2020. url:
https://cormandrostenreview. com/. [26] R. Jaafar et al.: Correlation Between 3790 Quantitative Polymerase Chain Reaction-Positives Samples and Positive Cell Cultures, Including 1941 Severe Acute Respiratory Syndrome Coronavirus 2 Isolates. Clin. Infect. Dis. 72 (2020), e921. pmid: 32986798. [27] F. M. Liotti et al.: Assessment of SARS-CoV-2 RNA Test Results Among Patients Who Recovered From COVID-19 With Prior Negative Results. JAMA internal medicine 181 (2020), 702–704. pmid: 33180119. [28] J. Bullard et al.: Predicting Infectious Severe Acute Respiratory Syndrome Coronavirus 2 From Diagnostic Samples. Clin. Infect. Dis. 71 (2020), 2663–2666. pmid: 32442256. [29] Anonymous: FDA briefing document: Pfizer-BioNTech COVID-19 Vaccine. 2020. url: https: //
www.fda.gov/media/144245/download. [30] Anonymous: Assessment report/Comirnaty. 2021. url:
European Medicines Agency | en/documents/assessment-report/comirnaty-epar-public-assessment-report_ en.pdf. [31] R. W. Frenck et al.: Safety, Immunogenicity, and Efficacy of the BNT162b2 Covid-19 Vaccine in Adolescents. N. Engl. J. Med. (2021). pmid: 34043894. [32] R. A. Campbell et al.: Comparison of the coagulopathies associated with COVID-19 and sepsis. Research and practice in thrombosis and haemostasis 5 (2021), e12525. pmid: 34027292. [33] G. H. Frydman et al.: The Potential Role of Coagulation Factor Xa in the Pathophysiology of COVID-19: A Role for Anticoagulants as Multimodal Therapeutic Agents. TH open : companion journal to thrombosis and haemostasis 4 (2020), e288–e299. pmid: 33043235. [34] Anonymous: SARS-CoV-2 mRNA Vaccine (BNT162, PF-07302048) 2.6.4 [Summary statement of the pharmacokinetic study] (Japanese). 2020. url:
https://www.pmda.go.jp/drugs/ 2021/P20210212001/672212000_30300AMX00231_I100_1.pdf. [35] I. C. Kourtis et al.: Peripherally administered nanoparticles target monocytic myeloid cells, secondary lymphoid organs and tumors in mice. PLoS One 8 (2013), e61646. pmid: 23626707. [36] C. Ye et al.: Co-delivery of GOLPH3 siRNA and gefitinib by cationic lipid-PLGA nanoparticles improves EGFR-targeted therapy for glioma. J. Mol. Med. Berl. 97 (2019), 1575–1588. pmid: 31673738. [37] R. Dal Magro et al.: ApoE-modified solid lipid nanoparticles: A feasible strategy to cross the blood-brain barrier. J. Control. Release 249 (2017), 103–110. pmid: 28153761. [38] R. B. Brown: Public health lessons learned from biases in coronavirus mortality overestimation. Disaster Med. Public Health Prep. (2020), 1–24. pmid: 32782048. [39] V. Furer et al.: Herpes zoster following BNT162b2 mRNA Covid-19 vaccination in patients with autoimmune inflammatory rheumatic diseases: a case series. Rheumatology (2021). pmid: 33848321. 22 [40] S. M. C. Tirado and K.-J. Yoon: Antibody-dependent enhancement of virus infection and disease. Viral immunology 16 (2003), 69–86. pmid: 12725690. [41] C.-T. Tseng et al.: Immunization with SARS coronavirus vaccines leads to pulmonary immunopathology on challenge with the SARS virus. PLoS One 7 (2012), e35421. pmid: 22536382. [42] F. Negro: Is antibody-dependent enhancement playing a role in COVID-19 pathogenesis? Swiss Med. Wkly. 150 (2020), w20249. pmid: 32298458. [43] J. A. Tetro: Is COVID-19 receiving ADE from other coronaviruses? Microbes and infection 22 (2020), 72–73. pmid: 32092539. [44] Y. Honda-Okubo et al.: Severe acute respiratory syndrome-associated coronavirus vaccines formulated with delta inulin adjuvants provide enhanced protection while ameliorating lung eosinophilic immunopathology. J. Virol. 89 (2015), 2995–3007. pmid: 2552050